Will I pass HPV to my baby during childbirth?

Posted in Uncategorized on Jun 01, 2010

My Fiance & I have HPV. Since I’ve been pregnant I had an outbreak of the warts.

Can I get them removed while pregnant?
Will I pass them on to my baby during childbirth?

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3 to “Will I pass HPV to my baby during childbirth?”


  1. Jenn says:

    No you will not pass it on to your baby. You are confusing genital warts with herpes. Herpes you CAN pass on, but not HPV. I would not recommend getting them removed while you are pregnant, in fact I don’t think you can do it w/o some kind of anesthesia or pain med. I can’t imagine removal is without pain. If you are truly concerned, talk with your OB for solutions. They know better than anyone else the risks involved. But no worries, your baby will be safe!! Good luck and congratulations on the pregnancy!!

  2. In rare cases a mother can transmit the virus to a child during child birth. The rare transmission affects the baby’s larynx or voice box…to learn more the Recurrent Respirator Papilloamvirus Foundation should be able to help you

    http://helpforgenitalwarts.com/genital-warts-pictures/"

    Your doctor should talk with you regarding your risk factor…and both you and your doctor will decide what is best for you and your child.

    In some women genital warts do grow during pregnancy and they are treatments that can be done during your pregnancy.

    I wish you and your baby well.

    Here is some information for you.

    Labor and Delivery
    Condylomata acuminata grow more rapidly during pregnancy and often regress spontaneously following delivery. Extensive condylomata acuminata may become secondarily infected, resulting in chorioamnionitis, preterm premature rupture of the fetal membranes, and episiotomy dehiscence. Condylomata are extremely vascular, and vaginal delivery may result in extensive hemorrhage, and rarely, in maternal death. Cryotherapy, or in the case of extensive lesions, laser therapy, may be effective in removing enough of the lesions to allow successful vaginal delivery. Debulking of lesions may also reduce the viral inoculum, and in turn, the risk of HPV infection, to the neonate at the time of delivery. Some experts recommend postponing “definitive” therapy until the mid third trimester to reduce the likelihood of recurrence prior to labor.
    Juvenile Laryngeal Pappilomatosis
    In 1871, MacKenzie noted the frequent association of skin warts and laryngeal papillomas. Two forms of laryngeal papillomas have been described. An adult form, nonaggressive and with solitary lesions and a male predilection, is often cured following a single operative procedure. Juvenile laryngeal papillomatosis (JLP), also called recurrent respiratory papillomatosis, on the other hand, is extremely aggressive and resistant to treatment, usually surgical. It typically involves the trachea, but may spread to the esophagus and bronchi, and rarely, to the lung where it actually destroys tissue, dramatically worsening the prognosis. Although rare, it is the most common benign tumor of the larynx.

    The incidence of JLP has been reported as 1:1500 live births. Because of its rarity, together with the relatively high prevalence of genital HPV, there has been some doubt about the cause of JLP. However, subtypes 6 and 11 of the HPV virus have been isolated from nearly all JLP lesions tested, and many experts believe that maternal-fetal transmission, is responsible for the lesions. Indeed, several retrospective studies have supported this mechanism of infection, with majorities of affected children born to mothers with documented HPV infection during pregnancy. One investigator found HPV DNA in nasotracheal aspirates of 48% of infants born to mothers with condylomata acuminata at the time of delivery. Prospective studies, however, have not been as supportive of maternal-fetal transmission. Among 44,000 infants followed for 7 years as part of the Collaborative Perinatal Project, none were found to have JLP. Another study, which found, retrospectively, that 5/9 infants with JLP were born to mothers with condylomata acuminata, found, prospectively, no cases of JLP in 31 infants born to women with overt HPV infection. Proponents of maternal-fetal transmission point out that the long latency period of 5 years or more for overt HPV infection of the larynx, and the small number of mother/infant pairs in the latter study, precluded any conclusion about the mechanism of infection in such a rare disease.

    Cesarean delivery has been proposed as a potential means of preventing JLP. However, although cesarean delivery is rare among infants and children with JLP, HPV DNA has been found in amniotic fluid before rupture of the fetal membranes and in oropharyngeal swabs of infants born by cesarean section. It is also unclear whether, as with genital HSV, recurrent lesions are less likely than primary ones to result in maternal-fetal transmission. The protective potential to the fetus/neonate of cesarean delivery probably does not exceed its potential morbidity to the mother. Most experts, including the CDC in its most recent treatment guidelines of STD’s, recommend cesarean delivery only for the usual obstetric and fetal indications, and for those with extensive lesions precluding a safe vaginal delivery for the mother.

    Of course, there are the medicolegal concerns regarding the association of maternal HPV and JLP. In fact, there has been a recent case favoring the mother of a child with JLP who alleged that her obstetrician did not adequately explain this very association to her. The rarity of JLP and its long incubation period have made it difficult for obstetricians to appreciate this manifestation of perinatal HPV. For those interested, information for parents of children with JLP can be obtained from the Recurrent Respiratory Papillomatosis Foundation, 50 Wesl

  3. well since youre pregnant and will HOPEFULLY be receiving prenatal care.. this is definitely a question to ask a trained medical professional. i wouldn’t listen to any joe shmoe on the interent.




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